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Within an interacting suggestions loop, CLOCK-BMAL1 complexes activate expression of nuclear receptors, REV-ERB α and RORα. Their protein goods feed-back to control BMAL1 by competitively binding retinoic acid-linked orphan receptor reaction components while in the BMAL1 promoter40. REV-ERBs repress the transcription of BMAL1, Whilst RORs activat